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Image from page 354 of "General surgical pathology and therapeutics, in fifty lectures : a text-book for students and physicians" (1871)

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Identifier: generalsurgicalp1871bill
Title: General surgical pathology and therapeutics, in fifty lectures : a text-book for students and physicians
Year: 1871 (1870s)
Authors: Billroth, Theodor, 1829-1894 Hackley, Charles E. (Charles Elihu), 1836-1925, tr
Subjects: Pathology, Surgical Therapeutics, Surgical Pathology, Surgical Therapeutics
Publisher: New York : D. Appleton and company
Contributing Library: Francis A. Countway Library of Medicine
Digitizing Sponsor: Open Knowledge Commons and Harvard Medical School


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Text Appearing Before Image:
oftraumatic fever is quite frequent. I explain it as follows: Immedi-ately after the injury the tissue of the edges of the wound was closedby infiltration of plastic matter; the third day this commenced tobreak down into pus, and to mingle with decomposed shreds of tissueon the surface of the wound, thus inducing a moderately extensiveinflammation of the amputation stump, with reabsorption of pus andother products of decomposition and inflammation; this reabsorptiongoes on till checked by some mechanical cause (diminished pressure, TRAUMATIC AND INFLAMMATORY FEVER. 331 thickening and partial closure of the vessels, etc.). In other cases,the fever begins the very day of the injury; we see this when bloodhas been enclosed between the flaps of the united wound and it hasrapidly decomposed; frequently, also, when operations have beendone in tissues infiltrated with the products of chronic infiltration.The following case (Fig. 61) may serve as an illustration of thissecond class: Fig. 61.

Text Appearing After Image:
Fever-curve after resection of a carious wrist, with great infiltration of the soft parts. Recovery. In infiltration of the tissue from chronic inflammation, the finerlymphatic capillaries may be contracted and to some extent closed,and hence, for some time, may not have carried off sufficient serumfrom the tissue, but the medium-sized lymphatic vessels, like the cor-responding veins, which in chronic inflammation have long been ex-posed to high pressure, are undoubtedly distended, perhaps evengaping, from rigidity of their walls; hence, if not quickly filled withfirm plastic infiltration from the start, they take up a good deal of thesecretion from the wound ; moreover, on the edges of wounds in mor-bidly-infiltrated tissue, mortification is particularly apt to occur. Thisexplanation of the late and early occurrence of traumatic fever ispurely hypothetical; but it is taken from and has been induced bynumerous observations. It might also be assumed that in one case 332 TRAUMATIC AND I


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Date: 2014-07-28 14:55:34



bookid:generalsurgicalp1871bill bookyear:1871 bookdecade:1870 bookcentury:1800 bookauthor:Billroth__Theodor__1829_1894 bookauthor:Hackley__Charles_E___Charles_Elihu___1836_1925__tr booksubject:Pathology__Surgical booksubject:Therapeutics__Surgical booksubject:Therapeutics bookpublisher:New_York___D__Appleton_and_company bookcontributor:Francis_A__Countway_Library_of_Medicine booksponsor:Open_Knowledge_Commons_and_Harvard_Medical_School bookleafnumber:354 bookcollection:medicalheritagelibrary bookcollection:francisacountwaylibrary bookcollection:americana

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